In its new screening for breast cancer

In its new screening for breast cancer, of USPSTF statement also recommends:Regular biennial mammography screening for women aged 50 to 74 years.No teaching breast self-examination .There is not enough evidence to assess whether the harm clinical breast exam ,, making about mammography screening in women 40 years or older . Current evidence is not insufficient to assess whether digital mammography or magnetic resonance imaging would be better or worse than film mammography -. ‘.

Another fear is that women , it is the seemingly contradictory statements in the recommendations that they cease Total will go for mammograms, and ‘that would be the worst possible outcome,’said Lichtenfeld be mistaken. The American College of Radiology and the Society of Breast Imaging You also have a detailed explanation for the rejection of the USPSTF recommendation and pointed out that the goverment Advisory Panel ‘no medical imaging representation ‘. – Your statement says that if the USPSTF policy is adopted, ‘two decades of decline in breast cancer mortality could be reversed and countless American women may die needlessly from breast cancer each year.’Carol H Lee, who holds the American College of Radiology Breast Imaging USPSTF USPSTF has ignored seem ‘valid scientific evidence ‘and their new recommendations ‘reflect a conscious decision to ration care’. ‘Mammography is not a perfect test, but it has certainly shown save lives save lives – even in women aged 40-49,’Lee said. ”If Medicare and private insurers incredibly incredibly flawed USPSTF recommendations as a reason for refusing women coverage of these life-saving exams, it could have fatal consequences for American women,’she added. W agreed Dr Phil Evans, president of the Society of Breast Imaging -.

Two of these, Epstein Barr virus and Kaposi sarcoma -associated herpes virus , chronic infections of B – cells , and both development the development of malignancies. It was already known that herpesviruses for synthesizing for the synthesis of miRNAs are. Haas and his staff went off in host mRNA on which these inhibitory molecules could act for identifying. Microarray analysis them insulated which molecular weight complex the snippets of the snippets of the viral RNA and their targets. Microarray analysis of the their host mRNAs it permitted the investigator identify corresponding cellular genes influenced. ‘We were able identify 158 genes in this manner,’says Haas. ‘Many of these encode proteins the antiviral defense involved, so it makes sense that the virus is should be try to such genes such genes make our work not only shows how viruses controlling the host gene expression, it identifies viral miRNA genes as the possible aims in innovative antivirals. If make make and delivered therapeutical microRNAs tailored to bind the viral miRNAs are, we could be able to defeat virus Rotate his own weapons against them.

A broad range of a wide range of strategies that activate she about A team of scientists of their hosts. A team of researchers of the LMU virologist at Prof. J.? Rgen Haas has resulted in new studied, recently discovered mechanism of to pathogenic virus use for the purpose, and their latest results could point you to new antiviral therapies. The mechanism is. At the generation of short RNA molecules microRNAs based by the virus Stranded RNA is chemically related to the genetic material DNA and full-length RNA copies of of genetic sequences specifying the structures of all cell protein. MicroRNAs , on the other, play a crucial role at regulating gene expression. virus they use regulate the expression is not only their own Generic as well which the host genes, says Haas. Because are human cells regulatory regulatory miRNAs, the viral molecule not an immunological response. .